At the end of the incubation time (DAT 120) the amount of 14CO2 reached 6.2-8.5 % of AR with radiolabelling in the pyrazolyl-ring and 5.2 % with radiolabelling in phenyl-ring. No other volatile compounds were detected. The portion of non-extractable residues (NER) steadily increased reaching its peaks between 38.9 and 69.9 % of AR at DAT 120 with radiolabelling in the pyrazolyl-ring and 64.0 % with radiolabelling in phenylring. A partitioning of the heterogeneous NER into humic acid, fulvic acid and humin like fractions was observed. The active substance fenpyrazamine degraded mostly to CO2 and bound residues, but small amounts of radioactivity were identified as metabolites S-2188-OH and S-2188-DC, though the concentrations never exceeded 5 % of AR. The unknowns represented maximum 6.7 % of AR. Undifferentiated regions were regions that did not form discrete peaks and accounted for up to 10.2 % of AR. Under the study conditions (20 ± 2 °C, moisture set at the mean of the water holding capacity values at pF 2 and pF 2.5) degradation of S-2188 was observed, reaching about 10 % of AR in soils PT102 and SK920191, and more than 30 % of AR in soils PT103 and SK15556090. During the study period, no major or minor metabolites could be identified in the tested soils. The metabolite S-2188-OH was identified at maximum 2.4 % of AR (DAT 30 in soil SK920191). The metabolite S-2188-DC was identified at maximum 2.6 % of AR (DAT 14 in soil SK920191). Regions with undifferentiated radioactivity accounted for up to 10.2 % of AR (in soil PT103). This radioactivity is considered to represent radioactivity from S-2188 that was well degraded, and had started to become incorporated into the soil organic matter. It was extracted from soil mainly in the acid extraction solvent rather than the initial neutral solvent. Further, there were up to 11 unknown degradation products for a total of 6.7 % of AR at any time in any soil. The maximum level of any single degradation product was 3.1 % of AR. Metabolic pathway: S-2188 was metabolised under aerobic conditions by cleavage of the thiocarbamate linkage to form S-2188- DC which was further metabolised by hydroxylation at the 4-position of the pyrazolyl ring and tautomerism to form S-2188-OH. Further degradation occurred through mineralisation to CO2 and formation of bound residues. Other possible pathways are the formation of S-2188-OH from S-2188 not involving S-2188-DC and pathways leading to the minor unidentified metabolites.