[methoxyiminotolyl-ring-UL-14C] fluoxastrobin and [pyrimidine-2-14C] fluoxastrobin. Fluoxastrobin was shown to be slowly degraded in soil under aerobic conditions to the final degradation product CO2. For the [methoxyiminotolyl-ring UL-14C] fluoxastrobin the following degradation was seen in four different soils. For [pyrimidine-2-14C] fluoxastrobin the following degradation was seen in two different soils. Mineralisation appears to be more rapid with the higher organic carbon (oc%) soils. Greater mineralisation of the pyrimidine label was apparent compared to the methoxyiminotolyl label (4-34% compared to 0.7-10% respectively for the Byromville loamy sand and the Hoefchen silt). Three metabolites were identified in the soil extracts along with the parent compound and 14CO2. These were the major metabolite HEC 5725-E-des-chlorophenyl (M48-E) and the minor metabolites HEC 5725-E-hydroxyphenyl (M02-E) and HEC 5725-amide (%AR) are as follows: M02-E at 4.5%; M38 at 1.4% and M48-E at 32.2%. Fluoxastrobin was mainly metabolised to M48-E by cleavage of the chlorophenyl moiety, whilst M02-E and M38 were formed by hydroxylation of the chlorophenyl moiety or cleavage of the dioxazin ring respectively.